"In the acute stage, cerebellar infarcts are first seen as hyperintense areas on DWI because of cytotoxic oedema with restricted diffusion, after which they soon become hyperintense on FLAIR and T2WI as well 9. Brain swelling peaks at about 3 days, after which infarcts diminish in size and diffusion (pseudo-)normalises after around 10 days (1-4 weeks) 9. Also around 10 days, (subacute) infarcts may become hard to identify due to a phenomenon known as “fogging” 9. In this stage, infarcts may also be seen as areas of parenchymal contrast enhancement, which appears around 1 week and may be visible up to 2 to 4 months after infarction. Finally, large and small cerebellar infarcts tend to heal with cavitation, leaving a CSF-filled cavity surrounded by a rim of gliosis 10."
"Many of the symptoms of cerebellar infarction are non-specific, such as nausea, vomiting, dizziness, unsteadiness, and headache, and the clinical diagnosis relies on a focussed neurological examination and a reasonable index of suspicion. Examination findings include incoordination, ataxia, and horizontal nystagmus. Patients may also present with altered conscious state or coma."
"Typical features of infarction such as early loss of grey-white differentiation, hypoattenuation and oedema, progressing to chronic encephalomalacia may be identified in the relevant vascular territories."
"In the acute stage, cerebellar infarcts are first seen as hyperintense areas on DWI because of cytotoxic oedema with restricted diffusion, after which they soon become hyperintense on FLAIR and T2WI as well 9. Brain swelling peaks at about 3 days, after which infarcts diminish in size and diffusion (pseudo-)normalises after around 10 days (1-4 weeks) 9. Also around 10 days, (subacute) infarcts may become hard to identify due to a phenomenon known as “fogging” 9. In this stage, infarcts may also be seen as areas of parenchymal contrast enhancement, which appears around 1 week and may be visible up to 2 to 4 months after infarction. Finally, large and small cerebellar infarcts tend to heal with cavitation, leaving a CSF-filled cavity surrounded by a rim of gliosis 10."