"risk of rupture is less than for typical AAA."
"intima: atherosclerosis, much like typical AAA"
"media: atrophy and loss of elastic tissue"
"adventitia: marked inflammatory thickening with abundant lymphocytes, plasma cells, and macrophages"
"T1: hypointense"
"T2: hyperintense"
"T1 C+ (Gd): shows enhancement"
"elevated ESR (90%)"
"The aetiology of the inflammatory changes is poorly understood and many theories exist. Some believe it is an abnormally severe inflammatory response to the atherosclerotic process in the aorta. Some believe it is an autoimmune disease which is supported by the elevated ESR found in most patients; frequent constitutional symptoms; increased frequency of autoantibodies and other autoimmune diseases; and the not-uncommon family history of inflammatory AAA. Approximately 30% of IgG4-related aortitis involves abdominal aorta 4. Others postulate an infectious aetiology."
"Definitive treatment of the aneurysm is performed via EVAR or open surgical repair. Repairing the aneurysm causes varying degrees of regression of the periaortic fibrosis in many patients. Surgery may be necessary if there is entrapment of retroperitoneal structures."
"intima: atherosclerosis, much like typical AAA"
"media: atrophy and loss of elastic tissue"
"adventitia: marked inflammatory thickening with abundant lymphocytes, plasma cells, and macrophages"
"T1: hypointense"
"T2: hyperintense"
"T1 C+ (Gd): shows enhancement"
Expected headings
"Risk factors"
"Inflammation and fibrosis extend into the periaortic tissue and may entrap adjacent retroperitoneal structures such as the ureters or duodenum. There is some overlap with retroperitoneal fibrosis."
"The aetiology of the inflammatory changes is poorly understood and many theories exist. Some believe it is an abnormally severe inflammatory response to the atherosclerotic process in the aorta. Some believe it is an autoimmune disease which is supported by the elevated ESR found in most patients; frequent constitutional symptoms; increased frequency of autoantibodies and other autoimmune diseases; and the not-uncommon family history of inflammatory AAA. Approximately 30% of IgG4-related aortitis involves abdominal aorta 4. Others postulate an infectious aetiology."
"The aetiology of the inflammatory changes is poorly understood and many theories exist. Some believe it is an abnormally severe inflammatory response to the atherosclerotic process in the aorta. Some believe it is an autoimmune disease which is supported by the elevated ESR found in most patients; frequent constitutional symptoms; increased frequency of autoantibodies and other autoimmune diseases; and the not-uncommon family history of inflammatory AAA. Approximately 30% of IgG4-related aortitis involves abdominal aorta 4. Others postulate an infectious aetiology."
"The aetiology of the inflammatory changes is poorly understood and many theories exist. Some believe it is an abnormally severe inflammatory response to the atherosclerotic process in the aorta. Some believe it is an autoimmune disease which is supported by the elevated ESR found in most patients; frequent constitutional symptoms; increased frequency of autoantibodies and other autoimmune diseases; and the not-uncommon family history of inflammatory AAA. Approximately 30% of IgG4-related aortitis involves abdominal aorta 4. Others postulate an infectious aetiology."