"reduced lung function of a mixed (obstructive and restrictive) pattern, with elevated residual volume 22, reduced forced expiratoy volume (FEV1), reduced forced vital capacity (FVC), reduced diffusing capacity for carbon monoxide (DLCO))"
"One case has been described (c.2011), presenting as a hypermetabolic pulmonary nodule on 18F-FDG-PET with associated hypermetabolic axillary lymphadenopathy 7."
"reduced lung function of a mixed (obstructive and restrictive) pattern, with elevated residual volume 22, reduced forced expiratoy volume (FEV1), reduced forced vital capacity (FVC), reduced diffusing capacity for carbon monoxide (DLCO))"
"One case has been described (c.2011), presenting as a hypermetabolic pulmonary nodule on 18F-FDG-PET with associated hypermetabolic axillary lymphadenopathy 7."
"One case has been described (c.2011), presenting as a hypermetabolic pulmonary nodule on 18F-FDG-PET with associated hypermetabolic axillary lymphadenopathy 7."
Expected headings
"Associations"
"Complications"
"reduced lung function of a mixed (obstructive and restrictive) pattern, with elevated residual volume 22, reduced forced expiratoy volume (FEV1), reduced forced vital capacity (FVC), reduced diffusing capacity for carbon monoxide (DLCO))"
"Pleuroparenchymal fibroelastosis usually shows a progressive clinical course and carries a poor prognosis. There is no specific therapy. Medical options with steroids, cyclophosphamide, azathioprine, N-acetylcysteine, azithromycin, sulfamethoxazole and trimethoprim may all be tried. Antifibrotics are ineffectual. In progressive cases, lung transplantation may be required."
"Pleuroparenchymal fibroelastosis usually shows a progressive clinical course and carries a poor prognosis. There is no specific therapy. Medical options with steroids, cyclophosphamide, azathioprine, N-acetylcysteine, azithromycin, sulfamethoxazole and trimethoprim may all be tried. Antifibrotics are ineffectual. In progressive cases, lung transplantation may be required."