"In the absence of trauma or recent instrumentation of the joint, septic arthritis is usually secondary to haematogenous seeding. Staphylococcus aureus is the most commonly isolated agent (>50% of cases) 4,6, and Streptococci spp. are common; both these organisms can cause rapid joint destruction. Other pathogens include Pseudomonas, Escherichia coli, and Serratia marcescens 10."
"T1: low signal within the subchondral bone"
"T2: thin rim of subchondral oedema; pericapsular oedema 10"
"T1 C+ (Gd): synovial enhancement reflecting synovitis; pericapsular enhancement 10"
"The incidence of septic arthritis varies between 2-20 cases per 100,000 population per year 4."
"Haemophilus influenzae was once a common causative agent of septic arthritis in children under the age of 2, but its incidence has significantly decreased due to vaccination 8,9. Gonococcal bacteraemia 9 is more common in sexually active individuals and responds well to appropriate antibiotics, usually with no sequelae."
Expected headings
"Signal characteristics"
"The diagnosis of joint sepsis is often considered straightforward. Patients often present with a painful joint, fever and purulent synovial fluid."
"In the absence of trauma or recent instrumentation of the joint, septic arthritis is usually secondary to haematogenous seeding. Staphylococcus aureus is the most commonly isolated agent (>50% of cases) 4,6, and Streptococci spp. are common; both these organisms can cause rapid joint destruction. Other pathogens include Pseudomonas, Escherichia coli, and Serratia marcescens 10."
"T2: thin rim of subchondral oedema; pericapsular oedema 10"
"T1 C+ (Gd): synovial enhancement reflecting synovitis; pericapsular enhancement 10"