"cardiac: bradyarrhythmias, shortened QT interval, elevated ST segment, first degree heart block"
"cardiac: bradyarrhythmias, shortened QT interval, elevated ST segment, first degree heart block"
"Markedly elevated serum levels of 25-hydroxyvitamin D (25(OH)D) are key to the pathogenesis of vitamin D toxicity. Historically it was thought that as 25-hydroxyvitamin D rose, the hypercalcaemia manifested itself in step, however recent work suggests that in most people normocalcaemia persists, even in the face of a severely elevated serum 25(OH)D 2. Dosing of up to 20,000 IU per day seems to be safe in adult men 1,3."
"Markedly elevated serum levels of 25-hydroxyvitamin D (25(OH)D) are key to the pathogenesis of vitamin D toxicity. Historically it was thought that as 25-hydroxyvitamin D rose, the hypercalcaemia manifested itself in step, however recent work suggests that in most people normocalcaemia persists, even in the face of a severely elevated serum 25(OH)D 2. Dosing of up to 20,000 IU per day seems to be safe in adult men 1,3."
"steroid administration reduces hypercalcaemia by decreasing GI absorption of calcium"
"Radiological appearances of vitamin D toxicity generally are due to the subsequent hypercalcaemia, e.g. nephrocalcinosis, peptic ulcer disease, and acute pancreatitis."
Expected headings
"Exogenous"
"Endogenous"
"neuropsychiatric: deficient mentation, delirium, malaise, sleepiness, depression, psychosis, and in extremis, stupor and coma"
"gastrointestinal: repeated emesis, abdominal pain, loss of appetite, polydipsia, constipation, peptic ulcers, pancreatitis"
"neuropsychiatric: deficient mentation, delirium, malaise, sleepiness, depression, psychosis, and in extremis, stupor and coma"