"type 1: no imaging abnormality"
"type 2: high T2"
"type 3: high T2 with enhancement"
"type 4: haemorrhage or infarction"
"usually characterised as well-defined focal filling defects within the dural venous sinuses (measuring 2–9 mm in diameter) 4"
Expected headings
"Risk factors"
"Staging severity"
"Complications"
"Superior sagittal sinus or the dominant transverse sinus thrombosis can affect the arachnoid granulation absorption of cerebrospinal fluid; consequent cerebral swelling may occur 4. The subsequent venous hypertension can lead to oedema and haemorrhage."
"a potential pitfall is interpreting the distal superior sagittal sinus as being hyperdense near the torcula herophili; it is important to appreciate that normal blood within the dural sinuses is usually of slightly increased density relative to brain parenchyma and that true hyperdensity is the key to recognising thrombosis"
"Conventional spin-echo sequences may demonstrate an absence of normal flow void on the dural sinuses. The clot acutely is isointense on T1 and hypointense on T2 (this can mimic a flow void), with subacute clot becoming hyperintense on T1. All the findings listed in the CT section are also seen on MRI. The most sensitive conventional MRI sequences for detecting the clot are susceptibility sequences, such as SWI or GRE. Overall, conventional MRI sequences, when combined, are very sensitive but relatively nonspecific for detecting dural venous sinus thrombosis. Contrast-enhanced 3D T1WI GRE is the most sensitive and specific MRI sequence for detecting DVST; MRV will demonstrate a lack of flow."
"Dural venous thromboses can result in parenchymal oedema and ischaemia in its watershed area; the severity of which can be graded as follows:"
"direct signs of a thrombus (e.g. dense clot sign, cord sign, empty delta sign)"