"T1: slightly hypointense"
"T2/FLAIR: hyperintense"
"DWI: restricted diffusion"
"T1 C+ (Gd): may enhance if acute (or early subacute)"
"The term was penned by Charles Miller Fisher (1913-2012) 4, a Canadian neurologist, who described "lacunes" (Latin: lake) of empty fluid within the brains of stroke victims post-mortem."
"The term was penned by Charles Miller Fisher (1913-2012) 4, a Canadian neurologist, who described "lacunes" (Latin: lake) of empty fluid within the brains of stroke victims post-mortem."
""Lacunar infarcts" may be a useful collective term, as used in this article, but it is vague and its use is discouraged by the Standards for Reporting Vascular Changes on Neuroimaging 2 (STRIVE-2) 14. Lacunar infarcts encompasses the following entities/definitions that are endorsed by STRIVE-2 14:"
""Lacunar infarcts" may be a useful collective term, as used in this article, but it is vague and its use is discouraged by the Standards for Reporting Vascular Changes on Neuroimaging 2 (STRIVE-2) 14. Lacunar infarcts encompasses the following entities/definitions that are endorsed by STRIVE-2 14:"
"STRIVE-2 also defines cortical cerebral microinfarct, a notably distinct entity to all that 'lacunar infarcts' encompasses, describing acute infarcts"
"STRIVE-2 also defines cortical cerebral microinfarct, a notably distinct entity to all that 'lacunar infarcts' encompasses, describing acute infarcts"
"Acute lacunar infarcts (i.e. recent small subcortical infarcts (RSSIs) or incidental subcortical DWI-positive lesions), by STRIVE-2 definition, are ≤20 mm and are caused by occlusion of small penetrating end-arteries 14, including the lenticulostriate, thalamoperforating, and pontine perforating arteries, and the recurrent artery of Heubner."
"STRIVE-2 also defines cortical cerebral microinfarct, a notably distinct entity to all that 'lacunar infarcts' encompasses, describing acute infarcts"
"STRIVE-2 also defines cortical cerebral microinfarct, a notably distinct entity to all that 'lacunar infarcts' encompasses, describing acute infarcts"
"They are mostly thought to result from in situ microatheroma formation or lipohyalinosis 2. When this occurs at the origin of a penetrating artery, this is known as branch atheromatous disease 7. However, lacunar infarcts can also be described as being caused by embolism 5. Indeed, some argue that it is important to investigate lacunar infarction for underlying embolism even though this may only be present in a minority of patients 6."
"Lacunar infarcts represent about 20-35% of all ischaemic strokes. Annual incidence rates range from 13-33 per 100,000 people. Men are slightly more affected than women, and risk increases with age, particularly over 55 years 9-11."
"i.e. chronic lacunar infarct, although this terminology is discouraged unless it is certain that the lacune is caused by a small vessel infarct and not another pathology resulting in cavitation (e.g. previous subcortical haemorrhage)"
"Acute lacunar infarcts (i.e. recent small subcortical infarcts (RSSIs) or incidental subcortical DWI-positive lesions), by STRIVE-2 definition, are ≤20 mm and are caused by occlusion of small penetrating end-arteries 14, including the lenticulostriate, thalamoperforating, and pontine perforating arteries, and the recurrent artery of Heubner."